Chronic inflammation is one of the most common and most overlooked drivers of dry eye disease and ocular surface breakdown, and addressing it directly is often the key to achieving improvement that other treatments alone haven't been able to deliver.
How Inflammation Affects the Ocular Surface
The ocular surface is a sensitive, highly regulated environment. When inflammation takes hold, it affects virtually every component of the system. Inflammatory mediators damage the goblet cells that produce the mucin layer of the tear film, disrupting tear stability at its foundation. They impair meibomian gland function, reducing the quality of the lipid layer that prevents tears from evaporating too quickly.
They alter the corneal epithelium, compromising its protective barrier function and increasing surface vulnerability. And they activate nerve pathways that contribute to the discomfort, light sensitivity, and foreign body sensation that make chronic dry eye so difficult to live with. Addressing inflammation isn't just one part of dry eye treatment; for many guests, it's the part that makes everything else work.





